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Cytokine crosstalk within the bone marrow microenvironment as a novel resistance mechanism of Bcl-2/Mcl-1 inhibitor resistance in AML

Conclusion Over a long period, insulin has been regarded as a compensatory hormone of IGF-1 in cancer development and progression

Pegylation extends that window by attaching a polyethylene glycol chain to the peptide backbone, slowing renal clearance and giving researchers a longer usable window for site-specific study designs without eliminating the localized character of the response